Regorafenib

證據等級: L5 預測適應症: 10

目錄

  1. Regorafenib
  2. Regorafenib: From Metastatic Colorectal Cancer to Liposarcoma
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. Denmark Market Information
    7. Cytotoxicity
    8. Safety Considerations
    9. Conclusion and Next Steps
    10. Disclaimer

## 藥師評估報告

Regorafenib: From Metastatic Colorectal Cancer to Liposarcoma

One-Sentence Summary

Regorafenib is an oral multikinase inhibitor originally developed for metastatic colorectal cancer, GIST and hepatocellular carcinoma. The TxGNN model predicts it may be effective for Liposarcoma, with 2 clinical trials and 9 publications available — however, the two completed Phase II randomised trials that specifically tested this indication both found no clinical benefit in liposarcoma patients, directly contradicting the model’s prediction.


Quick Overview

Item Content
Original Indication Metastatic colorectal cancer, GIST, hepatocellular carcinoma (per international product labeling; no Danish marketing authorisation on file to confirm locally)
Predicted New Indication Liposarcoma
TxGNN Prediction Score 99.76%
Evidence Level L2 (2 completed Phase II RCTs directly addressing this indication — but with negative results)
Denmark Market Status Not marketed
Number of Marketing Authorisations 0
Recommended Decision Hold

Why is This Prediction Reasonable?

Regorafenib is an oral multikinase inhibitor targeting angiogenic (VEGFR1-3, TIE2), stromal (PDGFR-β, FGFR) and oncogenic receptor tyrosine kinases (KIT, RET, RAF) (per literature evidence, PMID 30069758). Its original approval was built on this broad antiangiogenic/antiproliferative profile in metastatic colorectal cancer, GIST and hepatocellular carcinoma.

Soft tissue sarcomas, including liposarcoma, are highly vascularised tumours in which angiogenesis signalling plays a key role in tumour biology (per REGOSARC trial protocol, PMID 25884155). This provided the mechanistic rationale for testing regorafenib across multiple sarcoma subtypes, and it is this class-level plausibility that the TxGNN model appears to be capturing.

However, mechanistic plausibility did not translate into clinical benefit for this specific subtype. The REGOSARC trial (PMID 27751846) showed regorafenib improved outcomes in leiomyosarcoma and synovial sarcoma but explicitly did not show benefit in the liposarcoma cohort. The independent SARC024 trial (PMID 32701199) confirmed this finding in a treatment-refractory liposarcoma population and concluded that “routine use of regorafenib in this patient population” is not supported. This is a case where a strong knowledge-graph-based mechanistic signal is directly contradicted by dedicated randomised clinical trial data.


Clinical Trial Evidence

Trial Number Phase Status Enrollment Key Findings
NCT01900743 Phase 2 Completed 219 REGOSARC: international, randomised, double-blind, placebo-controlled trial of regorafenib in metastatic/unresectable soft tissue sarcoma after anthracycline failure; liposarcoma was one of five predefined cohorts
NCT02048371 Phase 2 Completed 131 SARC024: blanket protocol studying oral regorafenib across selected sarcoma subtypes, including a dedicated liposarcoma cohort

Literature Evidence

PMID Year Type Journal Key Findings
27751846 2016 RCT The Lancet. Oncology REGOSARC: regorafenib improved progression-free survival vs placebo in non-adipocytic soft tissue sarcoma, but not in liposarcoma
32701199 2020 RCT The Oncologist SARC024 liposarcoma cohort: results do not support routine use of regorafenib in treatment-refractory liposarcoma
29902612 2018 RCT (post-cross-over analysis) European Journal of Cancer Updated REGOSARC analysis confirms lack of efficacy in liposarcoma even after placebo-to-regorafenib cross-over
28295221 2017 RCT post-hoc analysis Cancer Q-TWiST analysis of REGOSARC; quality-adjusted clinical benefit concentrated in non-adipocytic cohorts
25884155 2015 Study protocol BMC Cancer REGOSARC trial design; rationale based on angiogenesis’s role in sarcoma biology
29931504 2018 Review Targeted Oncology Reviews regorafenib’s evolving role across soft tissue sarcoma subtypes, including liposarcoma
33290314 2021 Retrospective study Anti-Cancer Drugs Anlotinib in WDLS/DDLS; cites regorafenib as an approved TKI for non-adipocytic STS, not liposarcoma specifically
40975452 2025 Review Critical Reviews in Oncology/Hematology Reviews maintenance therapy strategies after first-line treatment of advanced STS
26266019 2015 Case report Rare Tumors Pazopanib case in Ewing sarcoma; cited as part of the rationale for including a liposarcoma arm in SARC024

Denmark Market Information

Regorafenib currently has no marketing authorisation on file in Denmark (0 authorisations recorded; market status: not marketed).


Cytotoxicity

Item Content
Cytotoxicity Classification Targeted therapy (oral multikinase inhibitor: VEGFR1-3, TIE2, PDGFR-β, FGFR, KIT, RET, RAF)
Myelosuppression Risk Low–Moderate — literature evidence for this drug class emphasises hand-foot skin reaction, hypertension and diarrhoea over myelosuppression (PMID 30069758); no direct haematologic toxicity data in this evidence pack
Emetogenicity Classification Low (typical for oral multikinase TKIs)
Monitoring Items Blood pressure (VEGFR-inhibition-related hypertension, PMID 36583425), liver function (hepatotoxicity risk across anti-angiogenic TKIs, PMID 23981115), skin examination for hand-foot skin reaction (PMID 23700287), CBC
Handling Protection Oral targeted anticancer agent — follow institutional oral antineoplastic handling protocols; local TFDA/SmPC-specific handling requirements are a data gap pending label review

Safety Considerations

Please refer to the approved Summary of Product Characteristics (SmPC) for safety information. TFDA-equivalent label warnings/contraindications and DDI data were not available in this evidence pack (flagged as a Blocking data gap — DG001 — preventing initial safety screening).


Conclusion and Next Steps

Decision: Hold

Rationale:

  • Regorafenib has no existing marketing authorisation in Denmark, and local SmPC safety data is a Blocking gap that prevents even an initial safety screen.
  • More importantly, the two completed Phase II RCTs that specifically tested regorafenib in liposarcoma (REGOSARC, SARC024) both found no clinical benefit in this subtype, directly contradicting the TxGNN model’s high prediction score. This is a case where the model’s knowledge-graph-level signal does not hold up against dedicated clinical trial evidence.

To proceed, the following is needed:

  • TFDA/SmPC warnings, contraindications and drug interaction data (currently Blocking gap, DG001)
  • Formal mechanism-of-action documentation (DrugBank MOA field, DG002)
  • If pursued further, reframe as a research question around why TxGNN scores this indication highly despite negative trial data (e.g., biomarker-selected subpopulations, combination regimens) rather than pursuing liposarcoma monotherapy directly
  • Confirm whether any EU/EMA centralised authorisation exists for regorafenib under its approved indications, which would affect off-label pathway feasibility in Denmark

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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